Sunday, June 4, 2023

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6 thoughts on “Specific Mitochondrial Respiratory Defects & Compensatory Changes in ME/CFS Patient Cells
  1. So can the seahorse instrument be used for diagnosis? And possibly to mitigate 2 day cpet for disability claims?

    Doctors tend to withdraw when you tell them you haven't worked for 3 years due to illness and need disability to stave off homelessness.

  2. The model is almost identical to the effects of an NPC1 transporter protein deficiency/defect. Which would explain why some people improve while sick with a cold or infection and why some people improve with lorazepam or clonazepam .(via tspo pathway utilization of cholesterol to form neurosteroids) .

    A trial of etifoxine (stresam) might prove to be helpful for symptom management and might elucidate an underlying cause.

  3. I have been lucky enough to have been taught by Professor Fisher this semester and he is such a great lecturer and teacher. I am so glad he is on the ME/CFS team. I struggled this entire semester at uni as I have become sicker from pushing myself, but the staff at La Trobe were very understanding. Thank you for all the work you all put into this study.

  4. Professor Fisher has clearly put a lot of thought & effort in to creating slides to make this information easier for non scientists to understand and his work has not being shown, rendering this excellent talk useless to this audience. Does anyone have a link to a similar talk by Professor Fisher that has his slides included?

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