Study, Notes, & Amendments: TBA
MY BOOKS, COURSES, VIDEO REQUESTS, & CONSULTING
– ‘Understanding Creatine’ Book: https://bit.ly/31GxkSN
– Hire Me: https://bit.ly/3dmUl2H
– Content Requests: https://bit.ly/2QOVHLX
SUBSCRIBE TO OTHER SOURCES OF MY CONTENT [FREE]
– Instagram (@Physionic_PhD): http://bit.ly/2OBFe7i
– Facebook (Physionic): http://bit.ly/2PlIOaB
– Twitter (@PhysionicPhD): http://bit.ly/2POhuBs
– Email List: http://bit.ly/2AXIzK6
PODCAST [FREE]
– Spotify: http://bit.ly/SpotifyPhysionic
– Apple Podcast: https://apple.co/2k6bpT7
– Google Podcast: http://bit.ly/GooglePhysionic
– Stitcher: http://bit.ly/StitcherPhysionic
– Other Podcast Platforms: http://bit.ly/2PThKz0
0:00 – Introduction
3:44 – Study Introduction
4:54 – Fat Fates in Cells (Background)
8:37 – Fat and Cancer
24:50 – Mechanisms of Fat Reliance by Cancer Cells
41:50 – Conclusions/Take Aways
Created with Biorender.
#cancer #foodismedicine #healthyfood
source
Ofcourse high ldl generales inflamation and this produce free radicals
How do I gey rid of Myc protein
This channel is better than university
WOW , THIS INFORMATION IS REALY INTERESTING. MUCH I HAVEN'T LEARNED ABOUT… YET 🙂
NOW, THAT I'M IN MY SENOR YEARS, CANCER, HAS BEEN ON MY MIND A LOT!
CAN'T WAIT TO DIVE DOWN THIS RABBIT HOLE WITH YOU.
AND AS USUAL, I CAN'T WAIT FOR YOUR NEXT PRESENTATION>
Super interesting topic, thank you for bringing this to us. Do you see interaction of the disease of cancer with cardio- or cerebro-vascular diseases? If a if a cell is stiffer because it's incorporated much more saturated fatty acid than normal and if these cancer cells are mobile in the blood, could their decreased fluidity cause issues with vascular health? And since it's spewing out cholesterol, does this not indicate there would be an effect on lipoproteins and ultimately plaques?
Never skip to conclusion 😉
Eye opening 😮😮
Has it been shown these MYC over expressing cancers are using the fat for energy, or is it primarily to build new cells?
As I understood, one of the hallmarks of cancer is damaged and dysfunctional mitochondria. This would limit their ability to burn fat for energy via oxphos, focusing primarily on anaerobic methods. Of course fat is a significant component of cell membranes, so they still need it.
This is important in treatment though. Some cancers may be better to starve, such as Seyfried is researching. If some cancers can properly use fat for fuel, this method will not work for all cancers.
Again a valuable break down of the nitty-gritty Nicolas, thank you.
Request: Keto genic diet on adhd. Saturated vs unsaturated if you can find any
Very interesting. What if the CPT1 inhibitors were programmed to only target the specific genetic profile of the individual cancer cells? Wouldn’t that prevent the damage to normal cells? And is that possible?
This is very interesting.
Much research I have seen to date advokes for a keto and low protein diet to improve cancer outcomes. Also much was found on combining this with intermittent fasting which was found to put a "protective shield" around healthy cells whilst making cancer cells more vulnerable to chemotherapy.
Yet another excellent presentation, holding my attention the entire time. In trying to integrate this information, I found I was missing quantification of factors. I desire the ability to extend cell biology operating principles to effects on larger biological structures.
An interesting new Eye-Opening from you today again…wow! Looking forward to the nutrition part. Thank you
It should be clear Content you submit and the way you present it
Unlike anything here on YouTube
There are a lot of opinions on the internet But very few research-backed truths Your channel is a kind of university But free And without end-of-semester tests…
These mechanisms are so interesting. Please go deeper if you’d like.