On October 13, we sat down to chat with Mel B. Feany about the molecular processes underlying the neuropathology of alpha-synuclein and Parkinson’s disease. We focused on her findings implicating the actin cytoskeleton as an intermediary in mitochondrial dysfunction and other cellular mechanisms that contribute to pathology and cell death.
Guest:
Mel B Feany, Department of Pathology, Division of Neuropathology, Brigham and Women’s Hospital and Harvard Medical School.
Participating:
Chris Gamblin, Department of Neuroscience, Developmental and Regenerative Biology, UTSA
Hyoung-gon Lee, Department of Neuroscience, Developmental and Regenerative Biology, UTSA
Host:
Charles Wilson, Department of Neuroscience, Developmental and Regenerative Biology, UTSA
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