Watch the full interview with Dr. Robert Lustig on YouTube ?? https://bit.ly/474drrobertlustig
Dr. Robert Lustig is a neuroendocrinologist, New York Times bestselling author, and Professor of Pediatric Endocrinology at the University of California, San Francisco. He’s authored 125 peer-reviewed articles and 73 reviews.
Rob has become a leading public health authority on the impact sugar has on fueling the diabetes, obesity and metabolic syndrome epidemics, and on addressing changes in the food environment to reverse these chronic diseases.
Today we’re discussing his latest book, Metabolical: The Lure and the Lies of Processed Food, Nutrition, and Modern Medicine.
If you’re addicted to sugar, you’re going to want to watch this!
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About the Podcast
Jesse Chappus has in-depth conversations with health and wellness leaders from around the world. Topics include lifestyle, nutrition, fitness, self-help, sleep, meditation, spirituality and so much more. Tune in weekly to take your health to the next level!
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Watch the full interview with Dr. Robert Lustig on YouTube ?? https://bit.ly/474drrobertlustig
Jesse You may have captured the BEST 25 Minutes of Dr.Robert Lustig.PERFECT
Please show me a meal plan thats is cheap and healthy
Molasse is filled with fructose
?
Wow, interesting.
yeah right, but when we put this to the test, saturated fat is the worst for fatty liver disease and insulin resistance which also translate into t2 diabetes
People reading medical literature when?
OBJECTIVE
Nonalcoholic fatty liver disease (i.e., increased intrahepatic triglyceride [IHTG] content), predisposes to type 2 diabetes and cardiovascular disease. Adipose tissue lipolysis and hepatic de novo lipogenesis (DNL) are the main pathways contributing to IHTG. We hypothesized that dietary macronutrient composition influences the pathways, mediators, and magnitude of weight gain-induced changes in IHTG.
RESEARCH DESIGN AND METHODS
We overfed 38 overweight subjects (age 48 ± 2 years, BMI 31 ± 1 kg/m2, liver fat 4.7 ± 0.9%) 1,000 extra kcal/day of saturated (SAT) or unsaturated (UNSAT) fat or simple sugars (CARB) for 3 weeks. We measured IHTG (1H-MRS), pathways contributing to IHTG (lipolysis ([2H5]glycerol) and DNL (2H2O) basally and during euglycemic hyperinsulinemia), insulin resistance, endotoxemia, plasma ceramides, and adipose tissue gene expression at 0 and 3 weeks.
RESULTS
Overfeeding SAT increased IHTG more (+55%) than UNSAT (+15%, P < 0.05). CARB increased IHTG (+33%) by stimulating DNL (+98%). SAT significantly increased while UNSAT decreased lipolysis. SAT induced insulin resistance and endotoxemia and significantly increased multiple plasma ceramides. The diets had distinct effects on adipose tissue gene expression.
CONCLUSIONS
Macronutrient composition of excess energy influences pathways of IHTG: CARB increases DNL, while SAT increases and UNSAT decreases lipolysis. SAT induced the greatest increase in IHTG, insulin resistance, and harmful ceramides. Decreased intakes of SAT could be beneficial in reducing IHTG and the associated risk of diabetes.
Does he ever speak on artificial sweeteners? How do they compare with fructose as far as inhibiting ATP and insulin resistance?
So why did God make fruit? And why was that the main source of nutrition in the garden of eden?