Mitochondrial dysfunction is a significant contributor to neurodegeneration…
Neurodegeneration refers to the progressive loss of structure/function of neurons which are specialized cells in the nervous system, which includes the brain, spinal cord, and peripheral nerves. Neurons are responsible for transmitting electrical and chemical signals throughout the body.
Common symptoms of neurodegeneration include:
Memory loss 😶🌫️
Cognitive decline 🧠
Motor dysfunction 😵💫
Behavioral changes 😩
Mitochondria are often referred to as the “powerhouses” 🔋of the cells because they generate energy in the form of adenosine triphosphate (ATP).
Several mechanisms link mitochondrial dysfunction to neurodegenerative diseases.
In addition to energy production, the mitochondria also play essential roles in calcium signaling, apoptosis (programmed cell death), and the regulation of reactive oxygen species (ROS) levels.
🪫Energy Deficits: Neurodegenerative diseases often exhibit impaired energy metabolism. Insufficient ATP production can result in decreased neuronal function and contribute to the progressive loss of neurons.
✴️ Oxidative Stress: Mitochondria produce ROS as a byproduct of ATP production. Mitochondrial dysfunction can lead to excessive ROS production which can cause oxidative stress, damaging cellular components, including DNA 🧬 , proteins, and lipids therefore causing neurodegeneration.
🦴Calcium Dysregulation: Mitochondria play a crucial role in regulating intracellular calcium levels. Excessive calcium influx into mitochondria can disrupt their function and trigger cell death pathways.
⚫️Apoptosis: Mitochondria are intimately involved in the regulation of apoptosis. Dysfunction in the apoptotic pathways can result in either excessive cell death or impaired removal of damaged cells, both of which can contribute to neurodegenerative processes.
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